Exogenous itaconate addition in apolipoprotein E-deficient (Apoe?/?) mice suppressed the initiation and development of AAA and downstream inflammatory protein though Irg1 deficiency reverted the impact of inhibitory. Overexpression of Keap1 or transferred Cys151S mutant Keap1 vector also abolished the activation of Nrf2 induced by itaconate.Anti-in

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Compounds Along with the identical molecular formula Compounds While using the similar skeleton Use this molecule within a composition searchWe utilized regular methodological methods envisioned by Cochrane. Two overview authors independently evaluated the search results against the choice criteria, abstracted data, and assessed risk of bias.The br